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. 2022 Feb 11;23(4):2012. doi: 10.3390/ijms23042012

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Nerve injury-induced expression of TSLP in ipsilateral DRG neurons. (A) The hindpaw-withdrawal threshold (g) was detected using the calibrated forceps test at day 3, 7, and 14 after CCI. The ipsilateral (ipsi) injured sides showed a decreased paw-withdrawal threshold as compared to that in the contralateral (contra) side and sham group after injury. Values are presented as means ± s.e.m. (n = 6 rats). * p < 0.05; ANOVA test. (B) Lumbar 4/5th DRG on both sides were dissected. TSLP proteins were measured by western blot analysis when β-actin was used as the loading control. At day 7 and day 14 after CCI, TSLP proteins were increased in DRG in the ipsilateral injured side as compared to those in the contralateral side and sham group. (C) Each band signal density was quantitated and normalized to that of its own β-actin in each side. Values are presented as means ± s.e.m. (n = 6 rats). * p < 0.05, compared to contra side and sham group, Student’s t test.