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. 2022 Feb 2;14(3):1307–1320. doi: 10.18632/aging.203873

Figure 6.

Figure 6

miR-29 inhibited proliferation in uterine leiomyoma cells (UtLMCs). (A) miR-29 mimics inhibited STAT-3, Cyclin D1, and c-Myc in vitro compared with the NC control, and MiR-29 inhibitors promoted the protein expression of STAT-3, Cyclin D1, and c-Myc in vitro compared with the anti-NC control. (B) miR-29 mimicking suppressed cell proliferation in UtLMCs in vitro compared with the NC control, and MiR-29 inhibitors promoted cell proliferation in UtLMCs in vitro compared with the anti-NC control. Data are presented as mean ± SE (*P < 0.05, **P < 0.01). UtLMCs: uterine leiomyoma cells; NC: negative control; STAT-3: signal transducer and activator of transcription 3.