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. 2022 Feb 11;23(4):1997. doi: 10.3390/ijms23041997

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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(A) Intrinsic disorder predisposition of human S100B protein in comparison with the disorder profiles of human S100 proteins capable of IFN-β binding, estimated using PONDR^® VSL2 algorithm (http://www.pondr.com/ accessed on 26 November 2021). The plot represents data for the sequences aligned using Clustal Omega [64], and breaks in the curves correspond to the alignment gaps. (B) Sequence logo representation of human S100B, S100A1, S100A4, S100A6, and S100P proteins generated by the WebLogo tool (http://weblogo.threeplusone.com/ accessed on 26 November 2021) [65,66].