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. 2022 Feb 25;5:173. doi: 10.1038/s42003-022-03118-0

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Representative images to document rescue of the phenotype following Compound 9 treatment. The same AT-1 sTg mouse is shown. b and c Western blot showing levels of acetylated-Atg9a (Ac-Atg9a) in ER preparations from liver. Representative blots are shown in (b) while quantitation of results is shown in (c). d Representative images to document rescue of the phenotype following Compound 19 treatment. The same AT-1 sTg mouse is shown. e and f Western blot showing levels of acetylated-Atg9a (Ac-Atg9a) in ER preparations from liver. Representative blots are shown in (e) while quantitation of results is shown in (f). Bars represent mean ± SD. *p < 0.05, **p < 0.005, ***p < 0.0005, ^#p < 0.0001 via ordinary two-way ANOVA plaque area percentage with Tukey’s multiple comparison test. Treatment started at 2-months of age.