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. 2022 Feb 25;29:10732748221076682. doi: 10.1177/10732748221076682

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© The Author(s) 2022

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 7. — Patients with FAT1/2/3/4 mutations showed better response to ICIs in Hellmann NSCLC cohort. (A) PFS Kaplan–Meier survival curves comparing the mutant FAT1/2/3/4 with wildtype FAT1/2/3/4. (B) Comparison of BOR rates between mutant FAT1/2/3/4 and wildtype FAT1/2/3/4. (C) Comparison of DCB rates between mutant FAT1/2/3/4 and wildtype FAT1/2/3/4.