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. 2022 Feb 25;119(9):e2118818119. doi: 10.1073/pnas.2118818119

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Copyright © 2022 the Author(s). Published by PNAS.

This article is distributed under Creative Commons Attribution-NonCommercial-NoDerivatives License 4.0 (CC BY-NC-ND).

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Fig. 5. — Kurarinone alleviated the increase of sEH activity and suppressed the GSK3β signaling pathway via stabilizing the level of EETs in MPTP-induced PD mice. (A) Effect of kurarinone on 8,9-EET; 11,12-EET; 14,15-EET; 8,9-DHET; 11,12-DHET; 14,15-DHET; 8,9-EET/8,9-DHET; 11,12-EET/11,12-DHET; and 14,15-EET/14,15-DHET in MPTP-induced PD mice. Data represent mean ± SEM, n = 6. The significance of difference was determined by one-way ANOVA followed by Tukey’s test. (B) Effects of kurarinone on p-GSK3β and GSK3β expression levels in MPTP-induced PD mice. (C) Quantitative data of phosphorylated GSK3β (p-GSK3β)/GSK3β. Data represent mean ± SEM, n = 6. The significance of difference was determined by one-way ANOVA followed by Tukey’s test.