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. 2022 Mar 5;20:113. doi: 10.1186/s12951-022-01288-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Cytoprotection effect of MoSe₂-PVP NPs in vitro. a Quantitative analysis of ROS levels in RAW264.7 cells (left: healthy cells; middle: H₂O₂-treated RAW264.7 cells; right: H₂O₂/MoSe₂-PVP NPs-treated 264.7 cells). b Quantitative analysis of ROS levels in RAW264.7 cells. c Viability of RAW264.7 cells after treatment with 500 µM H₂O₂ and different concentrations of MoSe₂-PVP NPs (H₂O₂ -: without the H₂O₂; H₂O₂ + : with the H₂O₂. MoS₂-PVP -: without the MoS₂-PVP). A histogram diagram (d) and a scatter diagram (e) of cell apoptosis and necrosis distribution in untreated and MoSe₂-PVP NPs-treated RAW264.7 cells. Representative graphics are shown, n = 3 independent experiments