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. 2022 Mar 7;11(1):12. doi: 10.1038/s41389-022-00386-7

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A IPA was employed to map the interaction network between molecules and pathways significantly related to PSMC2. B qPCR was conducted to measure the levels of PSMC2 and RPS15A mRNA in MGC-803 cells after PSMC2 overexpression or knockdown. C WB was performed to measure the levels of PSMC2 and RPS15A proteins in MGC-803 cells after PSMC2 overexpression or knockdown. D Immumohistochemical staining was conducted to evaluate the expression of RPS15A in the gastric cancer tissues and para-carcinoma tissues. E WB was employed to detect the expression of mTOR pathway-related proteins after PSMC2 knockdown. F The expression of mTOR pathway-related proteins in MGC-803 cells with RPS15A overexpression or RPS15A overexpression and PSMC2 knockdown. *P < 0.05, ***P < 0.001, compared with Control group. Error bars meant SD.