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. 2022 Mar 8;12:4058. doi: 10.1038/s41598-022-07918-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Haematoxylin and eosin-stained sections from representative areas of lung parenchyma infected with COVID-19. The microphotograph shows (A) diffuse alveolar damage with the hyaline membrane (exudative phase) (× 200). (B) Diffuse alveolar damage with organization (organizing phase) (× 200). (C) Organizing pneumonia with interalveolar capillaries filled with microthrombi (× 200). (D) Alveolar spaces are filled with neutrophilic infiltrate in a case of organizing DAD (not shown here) indicate superimposed acute bronchopneumonia (× 100). (E, F) Hyperplastic pneumocytes (E, arrow) and megakaryocytes (F, arrow) are seen in a case of DAD with acute bronchopneumonia (× 400). (G) Representative image of SARS-CoV2-stained tissue from COVID-19 patient. SARS-COV2 IHC shows cytoplasmic granular positivity in pneumocytes.