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. 2022 Jan 29;17:1. doi: 10.1186/s12263-022-00704-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Cumulative hazards stratified by genetic vitamin D exposure for pre-specified clinical endpoints in hypertensive-diabetic subjects. Cumulative hazards curves supplemented with data on events-free survival. A Vitamin D genetic risk score (GRS) predicted combined cardiovascular (CV) endpoints in hypertensive subjects: mean survival (Below median: 123.6 [95%CI 119.8 to 127.5] months, versus ≥ median: 131.8 [95%CI 128.8 to 134.9] months, log-rank = 13.5, P<0.001); B Vitamin D GRS predicted incident myocardial infarction (MI): mean survival (below median: 145.1 [95%CI 142.6 to 147.7 months], versus ≥ median: 149.2 [95%CI 147.4 to 151.0 months], log-rank = 8.6, P = 0.003)