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. 2022 Feb 28;49(4):55. doi: 10.3892/ijmm.2022.5111

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Copyright: © Lee et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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(A and C) Western blot analysis of HIF-1α in CA1 (A) and CA2/3 (C) of the NT/sham, NT/ischemia, HT/sham, and HT/ischemia groups at 0, 3, 6 and 12 h, 1, 2 and 5 days after TFI. (B and D) Relative density of the immunoblot bands is represented. Protein levels of HIF-1α normalized to β-actin in CA1 (B) and CA2/3 (D) ^*P<0.05 vs. NT/sham group at 0 h; ^†P<0.05 vs. pre-time point group; ^#P<0.05 vs. NT/ischemia group. The bars indicate the means ± SEM (n=5, respectively). HIF-1α, hypoxia-inducible factor 1α; NT, normothermia; HT, hyperthermia; TFI, transient forebrain ischemia.