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. 2022 Mar 9;20:125. doi: 10.1186/s12951-022-01318-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 11 — Effects of PEG-CZNPs on podocyte injury in the B6;129-Gla^tm1Kul/J mouse model of FD. A Representative confocal immunofluorescence microscopy images of synaptopodin (red), with DAPI counterstaining (blue), in control and α-GLA deficient B6;129-Gla^tm1Kul/J mice with or without PEG-CZNP exposure. B Quantification of the synaptopodin intensities using ImageJ software. Group1; wild type, normal saline injection, Group 2; wild type + PEG-CZNP injection, Group 3; B6;129-Gla^tm1Kul/J mice + normal saline injection and Group 4; B6;129-Gla^tm1Kul/J mice + PEG-CZNP injection. Data values represent the mean ± SD; *P < 0.05 versus Group 1; ^#P < 0.05 versus Group 3 alone