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. 2022 Feb 24;13:857869. doi: 10.3389/fphar.2022.857869

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Copyright © 2022 Zou, Chen, Li, Li, Wei, Li, Wang, Chen and Huang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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EcN-MT treatment reduce liver injury, fibrosis, oxidative stress in the liver of mice. Values are presented as means ± SD (n = 3). (A) Treatment of EcN-MT in mice with subchronic Cd exposure. (B) Cd concentrations in the liver assayed by ICP-MS. (C) HE staining image of liver tissue (200×). (D) Western bloting analysis of a-SMA expression in liver tissues. (E) The relative expressions of a-SMA were quantified by ImageJ. GAPDH was used as an internal control. The activity of (F) MDA, (G) CAT, (H) GSH, (I) SOD. *p < 0.05, **p < 0.01, ***p < 0.001.