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. 2022 Feb 24;14(5):966. doi: 10.3390/nu14050966

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Effect of high dietary AGEs during pregnancy and lactation on reproduction in offspring. Perinatal exposure to high dietary AGEs affects the growth of the offspring as well as the onset of puberty, estrous cyclicity, and ovarian follicular development, including ovulatory events. In addition, perinatal exposure to high dietary AGEs causes the lower expression of ovarian genes involved in folliculogenesis (Amh and its receptor Amhr2), and steroidogenesis (Cyp19a1, which is an aromatase enzyme responsible for conversion of testosterone to estradiol), as well as lower serum AMH levels, which are a marker of ovarian reserve [88]. Antimullerian hormone (AMH), anti-mullerian hormone receptor (anti-mullerian hormone receptor), messenger RNA (mRNA).