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. 2022 Jan 29;41(11):1622–1633. doi: 10.1038/s41388-022-02185-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A The mRNA level of METTL3 after METTL3 overexpression in HuCC-T1 and HCCC-9810 cells was confirmed by RT-qPCR. B The protein level of METTL3 after METTL3 overexpression in HuCC-T1 and HCCC-9810 cells was confirmed by Western blotting. C Cell growth curve of HuCC-T1 and HCCC-9810 cells transfected with METTL3-OE or Vector. D Apoptosis analysis of HuCC-T1 and HCCC-9810 cells transfected with METTL3-OE or Vector. E Cell cycle analysis of HuCC-T1 and HCCC-9810 cells transfected with METTL3-OE or Vector. F Cell migration ability and cell invasion ability of HuCC-T1 and HCCC-9810 cells transfected with METTL3-OE or Vector. The results are presented as mean ± SD of three independent experiments. *P < 0.05, **P < 0.01, ***P < 0.001, according to a Student’s t test.