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. 2022 Mar 10;5:219. doi: 10.1038/s42003-022-03161-x

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Fluorescence micrographs of structures formed by CD200^low-hTERT-shp53-PIK3CA^H1047R in vivo in NOG mice stained for human K17, (hK17), K5, α-SMA (green), human K19 (hK19), K8/18 (red), and nuclei (blue) show histology reminiscent of biphasic lesions with an outer layer of K17⁺, K5⁺, and α-SMA⁺ MEP cells (arrows), surrounding a core of hK19⁺/K5⁺/K8/18⁺ hyperplastic cells (scale bar, 100 μm).