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. 2022 Feb 25;12:848296. doi: 10.3389/fonc.2022.848296

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Copyright © 2022 Yao, Zhang and Chen

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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In nasopharyngeal carcinoma, FOXD3-AS1 promotes cell proliferation, apoptosis, invasion, migration and stemness by restraining the expression of miR-135a-5p or microRNA-185-3p and elevating the expression of FOXD3. Clearance of miR-30c modulates the expression of CTHRC1 and thus enhances the proliferative, invasive, and migratory abilities of MDA-MB-468 cells. LINC00707 also interacts with miR-206 to upregulate ER-α expression, which induces the proliferation and apoptosis of MCF-10AT cells.