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. 2022 Mar 14;79(3):188. doi: 10.1007/s00018-022-04214-4

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — DSK regulates sugar sensitivity and feeding in Drosophila. (a) The MP1/MP3 neurons receive nutrient signals of unknown origin. Upon feeding DSK is released and inhibits responsiveness of afferent gustatory receptor neurons (GRNs) that express the DSK receptor CCKL17D3, as well as the sugar receptor Gr64f and the gene takeout (to). (b) Scheme depicting the action of MP1/MP3 neurons after food intake. It is likely that a parallel pathway acts on unknown neurons (indicated by ?) to decrease sugar sensing. Panel b is based on Guo et al. [45]