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. 2022 Mar 14;13(3):234. doi: 10.1038/s41419-022-04681-4

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A Numbers of differentially expressed genes during P. aeruginosa infection following ALKBH5 knockdown. B Enriched pathways of differentially expressed genes during P. aeruginosa infection following ALKBH5 knockdown. C Numbers of differentially methylated genes during P. aeruginosa infection following ALKBH5 knockdown. D Enriched pathways of differentially methylated genes during P. aeruginosa infection following ALKBH5 knockdown. E Hypermethylated and hypomethylated genes following ALKBH5 knockdown among common differentially expressed and methylated genes between P. aeruginosa and HSV-1 infections, or P. aeruginosa infection and LPS treatment displayed by 2, 4, and 6 h P. aeruginosa post-infection. F Upregulated and downregulated genes following ALKBH5 knockdown among common differentially expressed and methylated genes between P. aeruginosa and HSV-1 infections, or P. aeruginosa infection and LPS treatment displayed by 2, 4, and 6 h P. aeruginosa post-infection. G Upregulated and downregulated genes following ALKBH5 knockdown among common differentially expressed and methylated genes between P. aeruginosa and HSV-1 infections, or P. aeruginosa infection and LPS treatment further displayed by differentially expressed and methylated genes at 2, 4, and 6 h P. aeruginosa post-infection.