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. 2022 Mar 2;12:860320. doi: 10.3389/fonc.2022.860320

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Copyright © 2022 Cyran and Zhitkovich

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Expression of heat shock genes across different types of human cancers. Shown are fold changes in gene expression of individual HSPs in cancers, which were calculated by dividing the number of transcripts per million in cancer tissue by that in the corresponding normal tissue. Transcript data were accessed through GEPIA (gepia.cancer-pku.cn) (25). Cancer types analyzed: bladder urothelial carcinoma, breast carcinoma, cervical squamous cell carcinoma and endocervical adenocarcinoma, colon adenocarcinoma, diffuse large B-cell lymphoma (DLBCL), esophageal carcinoma, glioblastoma multiforme (GBM), head and neck squamous cell carcinoma, renal clear cell carcinoma, acute myeloid leukemia (AML), hepatocellular carcinoma, lung adenocarcinoma (lung-adeno), lung squamous cell carcinoma (lung-SCC), ovarian serous cystadenocarcinoma, pancreatic adenocarcinoma, prostate adenocarcinoma, rectum adenocarcinoma, sarcoma, skin melanoma, stomach adenocarcinoma and uterine carcinosarcoma. The use of imperfectly matched cell populations as normal controls for hematological malignanies (bone marrow for DLBCL and blood for AML) potentially confounded patterns of normalized HSPs expression in these cancers.