Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2022 Mar 12;15:779–797. doi: 10.2147/DMSO.S349769

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2022 Xu et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

PMC Copyright notice

The relationship between bone turnover markers and diabetic ketoacidosis (DKA) conditions. (A) Many risk factors, such as missing insulin shots, infection, and unawareness of DM in individuals with newly diagnosed diabetes, can all lead to ketoacidosis. (B) Besides the factors that cause osteoporosis (OP), such as deficiency of insulin, abnormal estrogen after menopause in women, aging, and imbalance of the hypothalamus-pituitary-Thyroid (HPT) axis, diabetic ketoacidosis causes serum Ca²⁺, 25 (OH) D, N-MID to decrease while the bone-breaking marker, β-CTX, to increase.