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. 2021 Dec 15;126(6):865–873. doi: 10.1038/s41416-021-01619-z

Table 3.

Molecular characteristics of PCCRCs with likely biological cause versus DCRCs.

Features Biological PCCRCs (n = 94) DCRCs (n = 98) P value* FDR**
APC gene mutation 24/68 (35.3) 39/79 (49.4) 0.121
BRAF gene mutation 16/68 (23.5) 8/79 (10.1) 0.049
FBXW7 gene mutation 7/68 (10.3) 9/79 (11.4) 1.000
KIT gene mutation 13/68 (19.1) 18/79 (22.8) 0.733
KRAS gene mutation 19/68 (27.9) 24/79 (30.4) 0.887
PIK3CA gene mutation 11/68 (16.2) 13/79 (16.5) 1.000
PTEN gene mutation 10/68 (14.7) 6/79 (7.6) 0.265
SMAD4 gene mutation 4/68 (5.9) 9/79 (11.4) 0.378
TP53 gene mutation 23/68 (33.8) 38/79 (48.1) 0.113
Gain of chromosome 13q 37/78 (47.4) 60/88 (68.2) 0.179
Loss of chromosome 17p 29/78 (37.2) 42/88 (47.7) 0.051**
Loss of chromosome 18q 34/78 (43.6) 64/88 (72.7) 0.062
MSI 22/93 (23.7) 9/94 (9.6) 0.017
CIMP high profile 48/94 (51.1) 32/98 (32.7) 0.015

PCCRC post-colonoscopy colorectal cancer, DCRC detected colorectal cancer, FDR false detection rate, MSI microsatellite instability, CIMP CpG island methylator phenotype.

*P value < 0.05 considered significant; ** FDR < 0.2 considered significant.