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. 2021 Jun 21;43(1):405–422. doi: 10.3390/cimb43010033

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Protective effect of IPEE on the tau pathway in mice cerebral tissue. (a) Representative Western blots for the total protein and expression of TNF-α, p-JNK, p-Akt, p-GSK3β, p-tau, p-NF-κB and β-actin. Protein expression levels of TNF-α (b), p-JNK (c), p-Akt (d), p-GSK3β (e), p-tau (f), p-NF-κB (g), BAX (h) and cleaved caspase 3 (i) normalized to β-actin. Results shown are the mean ± SD (n = 3). Data shown were statistically considered at p < 0.05, and different small letters in the graph represent statistical differences.

Protective effect of IPEE on the tau pathway in mice cerebral tissue. (a) Representative Western blots for the total protein and expression of TNF-α, p-JNK, p-Akt, p-GSK3β, p-tau, p-NF-κB and β-actin. Protein expression levels of TNF-α (b), p-JNK (c), p-Akt (d), p-GSK3β (e), p-tau (f), p-NF-κB (g), BAX (h) and cleaved caspase 3 (i) normalized to β-actin. Results shown are the mean ± SD (n = 3). Data shown were statistically considered at p < 0.05, and different small letters in the graph represent statistical differences.