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. 2022 Jan 31;41(12):1679–1690. doi: 10.1038/s41388-022-02199-9

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — Pathway analyses of proteins purified from biotin-treated, ARIH1-miniTurboID transfected Hek293 cells. a Enriched biological processes from gene ontology and reactome pathways, identified using the Molecular Signature Database (MSigDB). b Immunoblot of biotin, hnRNP E1, UPF1, Cortactin, Filamin A (FlnA), Cand1 and Fasn in Hek293 cells either mock transfected or transfected with empty vector miniTurboID or ARIH1-miniTurboID and treated with or without biotin for 3 h.