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. 2022 Mar 19;22:124. doi: 10.1186/s12935-022-02544-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 11 — Genetic depletion of SLC1A5 promotes the proliferation and migration and inhibits the apoptosis of MGC-803 cell line. A Effect of knockdown of SLC1A5 expression in MGC-803 cell line after transfection with siRNA. B Cell proliferation assay was analyzed by the CCK-8 method each day for 4 days. C Transwell assay was employed to evaluate the migration effects of SLC1A5-deficient MGC-803 cell line. The cells were imaged at ×20 magnification. D Flow cytometry apoptosis assay showed the SLC1A5-deficient inhibits apoptosis rate in MGC-803 cell line. Data are presented as the mean ± SEM. Statistical significance was analyzed by ANOVA or Student’s t test. *p < 0.05, **p < 0.01, ***p < 0.001