Table 3.

Papers included in the summary of signaling cascades shown in Figure 3.

Article	Main findings
Lai et al. Redox Rep. 2018; 23(1):130-135. [54]	ROS induces proliferation and differentiation of Th17/Th1/Th22 cells.
Johansen et al. Br J Dermatol. 2005; 152(1):37-42. [57]	Activity of the MAPKs and ERK1/2 is increased in psoriatic skin.
Müller et al. Autophagy. 2020; 16(8):1380-1395. [59]	ROS are important mediators in IL23A secretion via NF-κB and MAPK pathways.
Zhou et al. Free Radic Biol Med. 2009; 47(7):891-905. [62]	Increased inflammation facilitates production of ROS and decreases already weakened antioxidative capacity
Srivastava et al. Indian J Dermatol Venereol Leprol. 2018; 84(1):39-44. [68]	Polymorphisms in the GST genes may result in increased production of ROS that could influence the pathogenesis of psoriasis.
Lee et al. J Invest Dermatol. 2013; 133(3):732-741. [70]	SOD deficiency resulted in more severe IL-23-mediated psoriasis-like skin inflammation.
Drewa et al. Med Sci Monit. 2002; 8(8):BR338-43. [71]	SOD and CAT activities were significantly lower in psoriatic patients.