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. Author manuscript; available in PMC: 2022 Mar 21.

Published in final edited form as: J Biophotonics. 2020 Nov 3;14(2):e202000359. doi: 10.1002/jbio.202000359

Figure 1A — High fluence LED-RL prevents cell cycle progression and is associated with arrest at G₀/G₁ phase in CRL-2617 fibroblasts at 18 hours post-irradiation (** p < 0.01).

Figure 1B Corresponding flow diagram demonstrating high fluence LED-RL is associated with G0/G1arrest of CRL-2617 fibroblasts at 18 hours post-irradiation.

Figure 1C High fluence LED-RL was associated with increased phosphorylated p53 expression in CRL-2617 fibroblasts at 18 hours post-irradiation. GAPDH served as a loading control.

Figure 1D High fluence LED-RL did not change baseline p53 expression in CRL-2617 fibroblasts at 18 hours post-irradiation. (.88 AG13145, .83 CRL-2617) GAPDH served as a loading control.

Figure 1A — High fluence LED-RL prevents cell cycle progression and is associated with arrest at G₀/G₁ phase in CRL-2617 fibroblasts at 18 hours post-irradiation (** p < 0.01).

Figure 1B Corresponding flow diagram demonstrating high fluence LED-RL is associated with G0/G1arrest of CRL-2617 fibroblasts at 18 hours post-irradiation.

Figure 1C High fluence LED-RL was associated with increased phosphorylated p53 expression in CRL-2617 fibroblasts at 18 hours post-irradiation. GAPDH served as a loading control.

Figure 1D High fluence LED-RL did not change baseline p53 expression in CRL-2617 fibroblasts at 18 hours post-irradiation. (.88 AG13145, .83 CRL-2617) GAPDH served as a loading control.