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Model of APAP sensitivity in writer and modification dysregulated Alkbh8Def livers. Alkbh8 plays a protective role against oxidative stress-induced APAP toxicity. Alkbh8Def mice experienced higher instances of oxidative stress, liver damage and overall decreased survival. The deficiency of Alkbh8 results in the dysregulation of the epitranscriptome and decreased selenoprotein expression.
