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. 2022 Jan 26;45(2):812–823. doi: 10.1007/s10753-021-01586-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Effect of RSV on liver function. Rat HF models were established with or without RSV treatment. Peripheral blood were collected after 3 months. A The chemical structure of RSV. B The survival rate of rats were analyzed (n = 12). Serum levels of ALT (C), AST (D), ALP (E), and γ-GGT (F) were tested using commercial kits. Data are mean ± SD from three independent experiments (n = 6 rats per group). For the high mortality of CCl₄-induced HF and the average survival rate is about 25%, more than 12 rats were used for each experiment to make sure that at least 3 rats were survived in the CCl₄ group 3 months after HF development. *p < 0.05, **p < 0.01, and ***p < 0.001.