Figure 1.

Summary of pathologic processes of long-noncoding RNAs (lncRNAs) in septic acute kidney injury (AKI). To summarize our findings, lncRNAs that primarily participate in four pathologic processes, which include inflammation, tubular cell injury, oxidative stress, and podocyte damage in septic AKI, are classified as indicated. In each category, the lncRNAs found to act as a promoting factor in the sepsis AKI are highlighted in red, while the lncRNAs found to act as protective factors are depicted in green, and the lncRNA with a dual effect are depicted in brown.