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. 2021 Sep 6;38(2):205–224. doi: 10.1007/s43188-021-00103-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Dysregulation of Wnt pathway and epigenetic chromatin modification genes in male mice exposed in utero to e-cig aerosols. Male mice exposed in utero to e-cig aerosol exhibited dysregulation of a number of genes associated with the Wnt signaling pathway and epigenetic chromatin modification at both PND5 and 11. N = 3–4 per group. Data are expressed as mean fold-change compared to respective in utero air control group. Data > ± 1.5-fold-change were considered significant