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. 2022 Mar 9;4(2):fcac055. doi: 10.1093/braincomms/fcac055

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© The Author(s) 2022. Published by Oxford University Press on behalf of the Guarantors of Brain.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Tau PET SUVR in the PCC is related to PCC-to-AG hypoconnectivity but not PCC-amPFC connectivity. (A) Increased tau PET signal in the left PCC is related to reduced functional connectivity between the left PCC and AG (r = −0.51, P = 0.01) in Aβ+ Alzheimer’s disease. (B) There was no relationship between tau PET signal in the left PCC and functional connectivity between the left PCC and amPFC (r = −0.29, P = 0.2). PCC, posterior cingulate cortex; AG, angular gyrus; amPFC, anterior medial prefrontal cortex; LH, left hemisphere. Functional connectivity units are Fisher’s z-scores. 95% confidence intervals are displayed. Results from the left hemisphere are shown here for illustrative purposes; complete results from both hemispheres are described in the text.