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. 2021 Dec 8;9(2):391–409. doi: 10.1007/s40744-021-00404-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial 4.0 International License, which permits any non-commercial use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc/4.0/.

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Fig. 1 — Fine-specificity ACPAs in patients who were anti-CCP2+ at baseline. a Correlation plot of ACPAs at baseline^a; b Change in ACPA specificity profiles from baseline to year 2 in the overall population of patients treated with abatacept or adalimumab; c Change in ACPA specificity profiles from baseline to year 2 in patients with MCR treated with abatacept or adalimumab. ^aVertical and horizontal axes list the ACPAs tested in the analysis; at the intersection between each row/column, spots of varying shades are presented depending on how correlated the two ACPAs were. ACPA anti-citrullinated protein antibody, anti-CCP2 anti-cyclic citrullinated peptide-2, FDR false discovery rate, MCR major clinical response. c adapted from Connolly SC, et al. EULAR Congress 2014; June 11–14, 2014; Paris France; poster FRI0039 (with permission of the authors)