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. 2022 Mar 30;14:14. doi: 10.1186/s13099-022-00488-y

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Synoptic representation of the genetic variations in the genome-wide resistance genes and linkage disequilibrium analysis. A The densities of total SNPs and Indels identified in the 112 H. pylori strains against H. pylori 26695 genome are overviewed. The numbers next to the rulers represent the number of SNPs and Indels per unit length of H. pylori 26695 genome. B The proportions of the strains with the nsSNPs and fsIndels present in the H. pylori genome-wide resistant genes in the resistant and susceptible categories of MTZ, CLA and LEV were analyzed, respectively. Only nsSNPs and fsIndels with significantly higher (red cells) or lower (blue cells) frequencies in the resistant categories of MTZ, CLA and LEV were displayed. *P < 0.05, **P < 0.01, ***P < 0.001. Conversely, grey cells indicate the variations not significantly associated with resistance or susceptibility to the antibiotics. Loci, genes, and variations are reported in rows and antibiotics in columns. The nature of the variations is color-coded on the left. The right panel represents the extent of linkage disequilibrium between variations