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. 2022 Jan 11;40(10):1081–1090. doi: 10.1200/JCO.21.01861

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© 2022 by American Society of Clinical Oncology

Licensed under the Creative Commons Attribution 4.0 License: https://creativecommons.org/licenses/by/4.0/

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FIG 1. — Genetic landscape and clinicopathologic characteristics of 131 pediatric thyroid cancers. Clinicopathologic characteristics include sex, age, histology, T status, N metastasis status, M, invasion, ETE, RAI therapy, and remission. The most frequent genetic alterations (fusion oncogenes and mutations) and their prevalence are shown. Genetic alterations were categorized into RET/NTRK fusions (RET and NTRK1/3 fusions), BRAF-mut (BRAF p.V600E), and RAS-mut (H-K-NRAS and PAX8/PPARG). cmvPTC, cribriform-morular variant papillary thyroid cancer; cPTC, classic papillary thyroid cancer; CSTP, CHOP Solid Tumor Panel; dsvPTC, diffuse sclerosing variant papillary thyroid cancer; ETE, extrathyroidal extension; FTC, follicular thyroid cancer; fvPTC, follicular variant papillary thyroid cancer; M, distant metastasis status; N, lymph node; RAI, radioactive iodine; svPTC, solid variant papillary thyroid cancer; T, tumor status; WLPTC, Warthin-like papillary thyroid cancer.