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. 2022 Mar 17;12:833582. doi: 10.3389/fonc.2022.833582

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Copyright © 2022 Rajthala, Parajuli, Dongre, Ljøkjel, Hoven, Kvalheim, Lybak, Neppelberg, Sapkota, Johannessen and Costea

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Fibroblasts’ proliferation and expression of CAF markers in monolayer culture following miR-138 modulation. Proliferation of (A) CAFs and (B) NOFs, (C) regulation of CCND1 transcript in CAFs, and (D–K) other transcripts related to the CAF-phenotype (TGFβ1, TGFBR2, and FAP) and EGFR in CAFs and NOFS 48 h post miR-138 modulation. * Significant; unpaired t-test p < 0.05. I138: inhibition of endogenous miR-138. M138: mimicking of miR-138 expression. Ictrl and MCtrl: respective controls for inhibitors and mimics. **p < 0.01, ***p < 0.001.