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. Author manuscript; available in PMC: 2022 Mar 31.
Published in final edited form as: Biochem Soc Trans. 2020 Feb 28;48(1):231–243. doi: 10.1042/BST20190597

Table 3.

Involvement of PCP in nonmalignant human lung disease

Gene (human
symbol)
Role in PCP Mouse mutant line Viability Lung phenotype References
Vangl1 (VANGL1) core PCP protein Vangl1CKO viable misoriented airway cilia Vladar et al. [60]
Vangl1Gt viable misoriented airway cilia Vladar et al. [60]
Vangl2 (VANGL2) core PCP protein Vangl2 Lp E18.5 lethal hypoplastic lungs with fewer branches; thickened interstitial mesenchyme; defective sacculation Yates et al. [21,36]
Vangl2 Lp E18.5 lethal misoriented airway cilia Vladar et al. [60]
Vangl2Lp heterozygote viable airspace enlargement; tissue damage reminiscent of emphysema Poobalasingam et al. [13]
Prickle2 (PRICKLE2) core PCP protein Prickle2 germline knockout viable misoriented airway cilia Vladar et al. [12]
Celsr1 (CELSR1) core PCP protein Celsr1 Crsh E18.5 lethal hypoplastic lungs with fewer branches; thickened interstitial mesenchyme; defective sacculation Yates et al. [21,36]
Flattop (CFAP126) putative PCP effector Fltp germline knockout viable ciliogenesis defect due to undocked basal bodies Gegg et al. [69]
Fuzzy (FUZ) putative PCP effector Fuz Gt1(neo) E18.5 lethal hypoplastic lungs Gray 2009 [81]
Ptk7 (PTK7) putative noncanonical co-receptor Chuzhoi E18.5 lethal hypoplastic lungs with fewer branches; thickened interstitial mesenchyme; defective sacculation Paudyal et al. [39]
Wntless (WLS) Wnt secretion regulator WlsShhCre (epithelial deletion) P0 lethal hypoplastic lungs with fewer branches; thickened interstitial mesenchyme; disrupted pulmonary endothelial differentiation Cornett et al. [45]
WlsDermo1Cre (mesenchymal deletion) E14.5 lethal lung abnormalities not observed at E14.5 Cornett et al. [45]
Wnt4 (WNT4) Wnt family ligand Wnt4 germline knockout E18.5 lethal hypoplastic lungs; defective cartilage formation Caprioli et al. [53]
Wnt5a (WNT5A) Wnt family ligand Wnt5a germline knockout P0 lethal short and wide trachea; abnormally branched distal airways; thickened interstitial mesenchyme; defective sacculation; endothelial cell and capillary disorganization Li et al. [43]
SpC-Wnt5a (epithelial overexpression) viable reduced epithelial branching Li et al. [46]
Wnt5aDermo1Cre (mesenchymal deletion) not reported abnormally polarized smooth muscle cells along trachea Kishimoto et al. [59]
Wnt5aECKO (endothelial deletion) viable Fewer pulmonary microvessels with decreased muscularization and pericyte coverage Yuan et al. [88]