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. 2022 Jan 14;13(2):2028–2043. doi: 10.1080/21655979.2021.2019872

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — ADIRF-AS1 knockdown restricts the malignancy of OS cells. (a) The interference efficiency of si-ADIRF-AS1#1 and si-ADIRF-AS1#2 in OS cells was validated by qRT-PCR. (b) Effects of ADIRF-AS1 deficiency on OS cell proliferation. (c) The apoptosis of OS cells treated with si-ADIRF-AS1 was examined by flow cytometric analysis. (d, e) The migratory and invasive properties of OS cells transfected with si-ADIRF-AS1 or si-NC. **P < 0.01.