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. 2022 Apr 1;17:146. doi: 10.1186/s13023-022-02295-9

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Short-wavelength fundus autofluorescence of three patients with mutations in TTLL5. Fundus autofluorescence images of P1 revealed sectoral atrophy involving the macula and the inferonasal retina with an irregular hyperautofluorescent ring surrounding the areas of atrophy (A). P2 exhibited a bull’s eye pattern of foveal hypoautofluorescence, more pronounced in the left than in the right eye (B). The foveal hypoautofluorescence secondary to macular pigment usually observed in normal eyes was not distinctly evident. P3 demonstrated a central bull’s eye pattern of hypoautofluorescence surrounded by a larger hyperautofluorescent ring extending further inferiorly than superiorly (C)