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. 2022 Jan 16;13(2):2308–2320. doi: 10.1080/21655979.2021.2024396

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 7. — Upregulation of SLIT3 neutralizes UBXN10-AS1’s tumor-suppressionin vitro. A. The expression of SLIT3 protein were determined by Western blots. B. CCK8 assays detected the proliferation capacity of indicated SW480 and SW620 cells. C. wound healing assay detected the migration capacity of indicated SW480 and SW620 cells. Empty vector, empty lentivirus vectors. UBXN10-AS1-OE, UBXN10-AS1-overexpressing lentivirus vectors. si-NC, siRNA negative control. *P < 0.05, **P < 0.001 vs. Empty vector; ^&&P < 0.001 vs. si-NC; ^#P < 0.05, ^##P < 0.001 vs. si-SLIT3+ UBXN10-AS1-OE.