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. 2022 Feb 14;13(3):5141–5151. doi: 10.1080/21655979.2022.2036914

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — RPN2 facilitated the tumorigenesis of LSCC cells. (a) Western blot assay for RPN2 protein levels in LSCC cells after overexpressing or silencing RPN2. (b) The viabilities of AMC-HN-8 cells transfected with RPN2 overexpressing or control pcDNA plasmid and TU212 cells transfected with RPN2 silencing or control sh-NC vector were analyzed by CCK-8 assay. (c) Cell migration was determined by Transwell analysis. (d) Protein levels of E-cadherin and Vimentin in LSCC cells were investigated by Western blot. *P < 0.05.