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. 2022 Mar 21;10:853127. doi: 10.3389/fcell.2022.853127

TABLE 1.

Clinical data in this study.

Age of seizure onset Sex Clinical manifestation Gene mutation Zygotic state Gene sequencing method Inheritance Reported Head MRI Treatment
4 years F GTCS, partial tonic, partial tonic–clonic c.5035C>T Het WES De novo Yes Normal VPA
R1679*
4 years F GTCS (10–15 min), partial tonic c.5035C>T Het WES De novo Yes Normal VPA
R1679*
40 days M GTCS, eyelid myoclonic, infantile spasm c.4173dup Het WES De novo Yes Less cerebral white matter PB → VPA, TPM, LTG, ACTH, KD
Q1392T*17
F Abnormal EEG, hyperactivity, mild intellectual disability c.4173dup Het WES De novo Yes Noncompliance None
Q1392T*17
5 years M GTCS (10 min, grumpy, talking to himself c.670C>T Het WES De novo No Abnormal (concrete is unknown) VPA
R224*
5 years M Partial tonic–clonic, only disturbances of consciousness, frequent seizures c.4012A>C Het epilepsy-targeted next-generation sequencing Unknown No Normal LEV, VPA, CBZ
K1338Q
3 years, 5 months F GTCS, myoclonic, partial tonic, partial tonic–clonic (20 min), frequent seizures c.2416dup Het WES De novo No Normal LEV, VPA, TPM, CLZ
R806Kfs*20
3 years, 1 months F GTCS, partial tonic–clonic (20 min), frequent seizures c.1727-1728insAT Het WES De novo No Small hippocampus LEV, VPA, TPM, PER, KD
E576Efs*13

GTCS, generalized tonic–clonic seizure; VPA, valproate; PB, phenobarbital; TPM, topiramate; LTG, lamotrigine; CBZ, carbamazepine; CLZ, clonazepam; PER, perampanel; ACTH, adrenocorticotropic hormone; KD, ketogenic diet; Het, heterozygous.