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. 2022 Apr 4;13(4):300. doi: 10.1038/s41419-022-04752-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — In young WAT, APSCs actively proliferate and differentiate to adipocytes. Resident immune cells keep in a relatively stable state. With advancing aging, APSCs gradually lose their developmental potential, leading to low adipogenesis. Aberrant immune cells and senescent cells accumulation drive the aging process of adipose. This figure was created in part with modified BioRender templates.