Skip to main content
. 2022 Apr 5;17(4):e0263113. doi: 10.1371/journal.pone.0263113

Fig 2. Proposed mechanism of oxidative stress in diabetic kidney disease.

Fig 2

O2, superoxide anion radical; H2O2, hydrogen peroxide; OH, hydroxyl radical; DNA, deoxyribonucleic acid; oxLDL, oxidized Low Density Lipoprotein; β2GPI, β-2-Glycoprotein-I; TGF-β1, transforming growth factor β1; 8-iso-PGF2α, 8-isoprostaglandin F2α; TP receptor, thromboxane-prostanoid receptor; GFR reduction, Glomerular Filtration Rate reduction.