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. 2022 Apr 5;5:310. doi: 10.1038/s42003-022-03266-3

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a, b Representative images of adipose tissue immunostained for PDGFRα, p15^INK4B, and γH2A.X. during wound healing in diabetic and non-diabetic patients. c, d Correlation between the percentage of p15^INK4B + or γH2A.X + cells in PDGFRα + cells and time post-wounding (n = 6 for each group). Correlations were examined statistically using Pearson’s correlation coefficient, and 95% confidence intervals are shown with translucent fill corresponding to each marker color.