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. 2022 Apr 5;8:164. doi: 10.1038/s41420-022-00925-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A The expression of SNHG12, LINC00665, and TTN-AS1 in GC and adjacent normal tissues examined using RT-qPCR (n = 69). B The relative level of SNHG12 in GC tissues from patients with GC of different clinical stages (I (n = 22); II and III (n = 47)) determined using RT-qPCR. C SNHG12 expression pattern in GC tissues with peritoneal metastasis (n = 31) and GC tissues without peritoneal metastasis (n = 38) from patients with GC detected by RT-qPCR. D The correlation between SNHG12 expression pattern and the overall survival of patients with GC analyzed by Kaplan–Meier survival curve. E The expression pattern of SNHG12 in serum-derived EVs in 10 healthy volunteers, 38 patients with non-metastatic GC, and 31 patients with metastatic GC determined by RT-qPCR. F SNHG12 expression pattern in normal human gastric mucosal cells GES-1 and GC cell lines HGC-27, NCI-N87, SNU-1, and SNU5 measured using RT-qPCR. G SNHG12 expression pattern in EVs isolated from each group of GC cells evaluated by RT-qPCR. *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001. The experiment was conducted three times independently.