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. 2022 Apr 5;4(1):e000267. doi: 10.1136/bmjno-2022-000267

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© Author(s) (or their employer(s)) 2022. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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Presumed mechanism of infarcts in penetrating artery territory. (A) Plaque in parent artery obstructing a branch. (B) Junctional plaque extending into the branch. (C) Microatheroma formed at the orifice of a branch. (D) Emboli from unstable microatheromatous plaque. (E) Fibrinoid degeneration or lipohyalinosis of the distal perforating artery. Y-YZ drew and created this figure with full permission. The authors confirm that this figure was not a reuse of previously published work.