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. 2022 Mar 28;25(5):186. doi: 10.3892/mmr.2022.12702

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Copyright: © Park et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Representative immunofluorescence confocal microscopy images. (A) miR-423 activates CaMKII phosphorylation in the control + miR-423 inhibitor group, Ang II group and Ang II + miR-423 inhibitor group but not the Ang II + miR-423 group. Total CaMKII (red fluorescence), p-CaMKII (green fluorescence) and DAPI (blue fluorescence). Magnification, ×400. (B) Quantitative analysis of p-CaMKII intensity. N=5 per group. *P<0.001. Data are presented as the mean ± SEM. Ang II, angiotensin II; CaMKII, calmodulin-dependent protein kinase II; CTL, control; inh., inhibitor; miR, microRNA; p-CaMKII, phosphorylated CaMKII.