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. 2021 Apr 2;38(2):259–272. doi: 10.1007/s10565-021-09600-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — STC2 participates in EMT and glycolysis pathway. a The expression level of STC2 detected by western blotting in nasopharyngeal carcinoma cell lines, including 6-10B, CNE1, 5-8F, C666-1, HK-1, and HONE-1 cells. b The validation of STC2 knockdown efficiency by western blotting. c Analysis of cell function regulated by RNA-seq. d qRT-PCR analysis of indicated genes in STC2 knockdown and its control cells. Data are shown as mean ± SD. Significance was calculated using Student’s t test. *p < 0.01