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. 2021 Apr 2;38(2):259–272. doi: 10.1007/s10565-021-09600-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — STC2 positively correlates with ITGB2/SOX6 and indicates a poor progression in clinical patients. a Representative images of IHC staining of STC2, ITGB2, and SOX6 in primary tissues and metastatic tissues. b The influence of STC2, ITGB2, and SOX6 on the overall survival in nasopharyngeal carcinoma. c Schematic model showing the role of the STC2/ITGB2/FAK/SOX6 signaling axis in the regulation of metastasis proliferation and chemical resistance